My lady’s doc wants to start her on Levothyroxine. My girl says she’s been fatigued for years, does not like the cold, and can’t get enough coffee.
She’s tried supplementing her diet with kelp for iodine, Brazil nuts for selenium, exercises daily, and takes a vitamin D supplement. Staples in her diet include steak, salmon, and a shit ton of avocados and mixed nuts.
She’s very fit and intelligent, but always tired. Her stress levels are moderate, and very rarely excessive. She does not have anxiety or depression. She sleeps well but always feels like she could use more.
Somebody please read pill me on this low thyroid business. Is it likely that she truly needs to take an exognous thyroid hormone for the rest of her life? I want to put babies in this white woman, but what the hell is going on with the thyroid?
How is this so common? Is this what God intended. Or is the medical establishment hiding some iatrogenic cause for “Hashimoto’s autoimmune thyroiditis”, or is there something in the food supply?
Please help me out if you have some pearls on this one goats, and merry Christmas.
Low Copper - Copper stimulates the production of the thyroxine hormone T4 and prevents over absorption of T4 in the blood cells by controlling the body's calcium levels.
Low magnesium- We need magnesium to convert the inactive thyroid hormone T4 into the active thyroid hormone T3. Without this conversion, cells do not receive the more potent form of thyroid hormone. Therefore, when magnesium levels are low, our thyroid is not able to function correctly.
Low Vitamin B6 (as pyridoxal 5 phosphate) B6 lowers prolactin and TSH. This can be because it lowers excess TRH, which stimulates the release of TSH and prolactin.
Low Zinc, Zinc is involved in the synthesis of TSH (since it participates in the synthesis of TRH. Zinc is involved in the synthesis of thyroid hormones. Zinc supplementation has been shown to increase T3.
Low Iron Low iron can reduce TPO activity and can increase rT3 deiodination, leading to thyroid hormone metabolism by inactivating pathway. Low iron can lead to inefficient erythropoiesis, consequently causing a decrease in oxygen transport to tissues. Oxygen is crucial for various enzymatic reactions (including thyroid hormone synthesis) Low iron can result in low T4 and T3 levels.
cj71 1 points 2.4 years ago
Could be many things.
Low Copper -
Copper stimulates the production of the thyroxine hormone T4 and prevents over absorption of T4 in the blood cells by controlling the body's calcium levels.
Low magnesium-
We need magnesium to convert the inactive thyroid hormone T4 into the active thyroid hormone T3. Without this conversion, cells do not receive the more potent form of thyroid hormone. Therefore, when magnesium levels are low, our thyroid is not able to function correctly.
Low Vitamin B6 (as pyridoxal 5 phosphate)
B6 lowers prolactin and TSH. This can be because it lowers excess TRH, which stimulates the release of TSH and prolactin.
Low Zinc,
Zinc is involved in the synthesis of TSH (since it participates in the synthesis of TRH. Zinc is involved in the synthesis of thyroid hormones. Zinc supplementation has been shown to increase T3.
Low Iron
Low iron can reduce TPO activity
and can increase rT3 deiodination, leading to thyroid hormone metabolism by inactivating pathway. Low iron can lead to inefficient erythropoiesis, consequently causing a decrease in oxygen transport to tissues. Oxygen is crucial for various enzymatic reactions (including thyroid hormone synthesis)
Low iron can result in low T4 and T3 levels.
Avoid calorie deficits.
Avoid plastics.
Manage stress.