Don’t know if you saw my other (long) comment. I do believe the a very sizeable portion of homosexual/lgbt disorders do have some sort of involvement of parasites or other contagions. Perhaps the majority. This is clearly what is happening with male cicadas infected with massaspora. Potentially affecting the transcription of male and female hormones. Who knows. But we know that parasites, fungi, etc. can program host with zombie like behaviors to promote spread. We know what it looks like in a cicada. What does it look like in a person? We need a very objective behavioralist interpretation. Psychology is full of these irrational psycho-therapy jackasses. There’s no proper science done in the field at all.
Ive been trying to think of a lifecycle that makes sense with respect to this theoretical parasite. There seem to be multiple subtypes of gay men? Are we looking at multiple opportunistic species? Or are different “types” of gays just different stages of the parasite’s lifecycle? Are bottoms and tops possibly people infected at different times in their lives (in utero or childhood, vs adulthood?) Do they represent people with active infections and people that are in remission? What role to bi-sexuals play? Infecting females?
Look at patterns in places like India and Thailand. First note the climate. There are no lady boys in Norway. (There are a few, but they haven’t been a thing for centuries). There may be some environmental conditons that favor this contagion.
If a Thai lady boy is part of the lifecycle of some fungus, for example, he could spread it to a “heterosexual” man looking for casual sex from a prostitute. This adult male, once infected, could pass the contagion to his wife and she could pass the contagion to her child in utero. This would result in a new lady boy, as the active infection would effect neurology. Even if this new lady boy was not infectious throughout his childhood, he would seek reinfection when he reached puberty (butt seks) and then become a carrier. Maybe this parasite effects brain structures of males in utero so that even if the infection is eliminated by the immune system, the infected person keeps seeking out re-infection from bum sex. Maybe that is the trick of this organism.
PostWallHelena 0 points 3.6 years ago
Don’t know if you saw my other (long) comment. I do believe the a very sizeable portion of homosexual/lgbt disorders do have some sort of involvement of parasites or other contagions. Perhaps the majority. This is clearly what is happening with male cicadas infected with massaspora. Potentially affecting the transcription of male and female hormones. Who knows. But we know that parasites, fungi, etc. can program host with zombie like behaviors to promote spread. We know what it looks like in a cicada. What does it look like in a person? We need a very objective behavioralist interpretation. Psychology is full of these irrational psycho-therapy jackasses. There’s no proper science done in the field at all.
Ive been trying to think of a lifecycle that makes sense with respect to this theoretical parasite. There seem to be multiple subtypes of gay men? Are we looking at multiple opportunistic species? Or are different “types” of gays just different stages of the parasite’s lifecycle? Are bottoms and tops possibly people infected at different times in their lives (in utero or childhood, vs adulthood?) Do they represent people with active infections and people that are in remission? What role to bi-sexuals play? Infecting females?
Look at patterns in places like India and Thailand. First note the climate. There are no lady boys in Norway. (There are a few, but they haven’t been a thing for centuries). There may be some environmental conditons that favor this contagion.
If a Thai lady boy is part of the lifecycle of some fungus, for example, he could spread it to a “heterosexual” man looking for casual sex from a prostitute. This adult male, once infected, could pass the contagion to his wife and she could pass the contagion to her child in utero. This would result in a new lady boy, as the active infection would effect neurology. Even if this new lady boy was not infectious throughout his childhood, he would seek reinfection when he reached puberty (butt seks) and then become a carrier. Maybe this parasite effects brain structures of males in utero so that even if the infection is eliminated by the immune system, the infected person keeps seeking out re-infection from bum sex. Maybe that is the trick of this organism.